A meta-analysis shows use of hormonal contraception, compared with nonhormonal or no contraception, ups the risk of HIV infection in sub-Saharan Africa.
High doses of vitamin D, which is an immune booster, aren't protective against bacterial vaginosis in women at high risk for STDs.
A pseudo-tampon that delivers an antiretroviral drug is now being studied as a form of pre-exposure prophylaxis for women at high risk for HIV infection.
Discitis, a bacterial infection within the vertebral disc, typically results from seeding during an episode of bacteremia.
Time to virologic failure among black patients on antiretroviral therapy was significantly shorter than for whites.
Over the past 4 decades, our understanding of the role of elevated cholesterol in cardiovascular disease (CVD) has undergone radical change. During that time, we have moved from a belief that cholesterol does not matter and that atherosclerosis is an irreversible process to a strong conviction that treating elevated cholesterol, especially elevated low-density lipoprotein cholesterol (LDL-C), can slow and perhaps halt the progression of atherosclerosis. But it has been a slow process for several reasons. In the 1960s, the Framingham investigators demonstrated that elevated serum cholesterol is a risk factor for CVD.1